docs: record oral B recovery boundary

5 files changed, 114 insertions, 44 deletions

diff --git a/PAPER_PLAN.md b/PAPER_PLAN.md
index 04f1d2f..290ac30 100644
--- a/PAPER_PLAN.md
+++ b/PAPER_PLAN.md
@@ -72,17 +72,16 @@ not imply that no-traffic scaling establishes the necessity of residualization.
    preservation, alignment, query/MAC/memory cost, and direct-versus-amortized
    causal feedback.
 4. **Scientific negatives.** Broad top-down traffic, useful-depth learned
-   vectorization, the Harnett desired-velocity signature, and full standard
-   ResNet stability fail their frozen gates, defining the method's actual
-   boundary.
+   vectorization, the Harnett desired-velocity/full population signature, and
+   the original learned-vectorizer ResNet branch fail their frozen gates,
+   defining the method's actual boundary.
 5. **Standard scale.** D3 passes all frozen checks at `91.18%` validation on
-   ResNet-20 versus BP's `91.62%`, but it is one seed.  Do not promote this to
-   a robustness or depth-scaling claim until the already frozen paired
-   five-seed D4 confirmation and a separate depth panel pass.
-   The separate dynamic-depth recovery in `ORAL_A_RECOVERY.md` is the only
-   permitted depth panel: after oral-B passes, it reuses D4's ResNet-20 cells
-   and adds BP/DFA/clean-KP/dynamic ResNet-32/56 comparisons under a frozen
-   positive-depth-benefit and cost gate.
+   ResNet-20 versus BP's `91.62%`; the independent paired D4 panel then reaches
+   `91.584%` mean test accuracy versus clean KP's `91.388%`. This supports
+   ResNet-20 robustness, not a positive depth-scaling claim. The separately
+   frozen dynamic-depth panel in `ORAL_A_RECOVERY.md` was the only permitted
+   route to the latter, but its oral-B R2 prerequisite fails, so no new
+   ResNet-32/56 endpoint is opened.
 
 ## Figure order in the manuscript
 
@@ -155,8 +154,10 @@ numbering.
   lesion; desired velocity is unsupported.
 - Keep the original failed screen intact. The separately frozen recovery
   factorizes learned causal role from within-episode performance innovation;
-  report it only if both its D4-gated development screen and untouched
-  6-task-by-5-model confirmation pass.
+  its D4-gated development screen passes, but the untouched 6-task-by-5-model
+  confirmation fails the joint population-vectorization/longitudinal gate.
+  Report the learning, lesion, sign-inversion, and velocity positives as a
+  bounded diagnostic, not as a passed oral-B claim.
 - Even a complete recovery pass supports innovation-guided plasticity, not
   online neural control: the recovery fixes `kappa=0` by construction.
 
@@ -177,7 +178,7 @@ numbering.
 | “Depth does not help this task.” | Claim says preservation; C2 reports the learned-vectorizer failure; Oral-A A3 is retained as a failed standard-scale test | Fatal to a broad scaling claim; the paper must remain mechanism-focused |
 | “Local methods hide enormous extra work.” | Logical queries, MACs, peak memory, wall time, C3 confirmation, EP/native protocols | Hardware implementations are not all equally optimized |
 | “Weak baselines define the win.” | BP/FA/DFA/direct NP/FF/PEPITA/EP plus native BurstCCN and Dual Prop | Native reproductions are one seed and method-native, not equal compute |
-| “Harnett already proves the biological story.” | Original oral-B negative result plus the separately frozen role/velocity recovery and untouched 30-record confirmation | No biological claim changes unless the recovery passes; `kappa=0` cannot support desired velocity |
+| “Harnett already proves the biological story.” | Original oral-B negative result plus the separately frozen role/velocity recovery and untouched 30-record confirmation | Recovery fixes learning and sign but fails the joint outcome/longitudinal gate; `kappa=0` cannot support desired velocity |
 | “The fast controller is just an unreported contrastive phase.” | Paired neutral observation count and elementwise/wall cost are explicit; no task-nudged state, loss query, or reverse pass is used | The instruction-off microphase is a real assumption and weakens the single-phase biological claim |
 | “The gates were selected after results.” | Git-frozen protocols, untouched confirmation seeds, failed branches retained | Early inherited pilots predate the strict boundary and must remain labeled |
 
@@ -276,6 +277,10 @@ ResNet-20 with an inherited zero-query reciprocal credit path.
 The paper must still attribute reciprocal KP plasticity to prior work, count
 the paired neutral microphase and elementwise work, and retain the failed
 arbitrary-top-down, desired-velocity, online-control, and earlier unstable
-branches.  D4 alone cannot support positive added-depth utility.  The next
-paper-changing gate is oral-B R1/R2; only its separately frozen confirmation
-can raise 7 to 8 and unlock the standard ResNet-20/32/56 oral-A panel.
+branches.  D4 alone cannot support positive added-depth utility. Oral-B R1
+selects `eta=0.1` with 98.05% worst-task development success, but the untouched
+R2 confirmation fails the broader outcome-vectorization and longitudinal
+signature checks despite 99.53% mean success and 30/30 positive sign
+inversions. The score therefore remains 7 and the frozen
+ResNet-20/32/56 oral-A panel stays closed. Do not repair thresholds or route
+around the failed prerequisite.
diff --git a/README.md b/README.md
index 0b712c4..767b425 100644
--- a/README.md
+++ b/README.md
@@ -91,13 +91,22 @@ and scaling behavior. See `NOVELTY.md` for the exact prior-art boundary.
 
 The broad endogenous-traffic gate and the Harnett desired-velocity/online-control
 screen failed. Those results are retained and explicitly constrain the paper:
-SDIL does not currently explain the reported error-derivative signature, and
-the residual mechanism is supported for traffic predictable from the chosen
-somatic statistic rather than arbitrary top-down context. `ROADMAP.md` and
-`ORAL_B.md` contain the frozen gates and complete negative branches.
-A mechanics-only recovery now separates perturbation-learned causal role from
-within-episode performance velocity; it passes its algebraic checks but has no
-task endpoint and does not yet weaken this limitation.
+SDIL does not currently explain the complete reported dendritic population
+signature or online control, and the residual mechanism is supported for
+traffic predictable from the chosen somatic statistic rather than arbitrary
+top-down context. `ROADMAP.md` and `ORAL_B.md` contain the frozen gates and
+complete negative branches.
+A temporal-difference recovery separates perturbation-learned causal role from
+within-episode performance velocity. Its development gate selects `eta=0.1`
+with 98.05% worst-task success, and its untouched 30-record confirmation
+retains 99.53% mean success, positive sign inversion in all 30 records, and a
+0.639 velocity-over-error correlation advantage. The complete R2 gate
+nevertheless fails: residual outcome decoding is only 47.33%, residuals trail
+soma by 4.32 points, and the longitudinal correlation is `-0.013`. Thus the
+recovery sharpens the boundary--causal-role temporal-difference plasticity
+works in this synthetic task, but the broader Harnett-like population
+vectorization signature is not established. The strict score remains 7/10
+and the oral-A depth panel stays sealed.
 
 ## Publication-facing artifacts
 
diff --git a/RESULTS.md b/RESULTS.md
index 436740f..c511d1e 100644
--- a/RESULTS.md
+++ b/RESULTS.md
@@ -184,6 +184,50 @@ learning, outcome decoding, and residual decorrelation do not imply the
 cell-role-specific error-derivative signature observed by Harnett and
 colleagues.
 
+## Temporal-difference oral-B recovery (R1/R2)
+
+The separately frozen recovery changes the structural target rather than
+reinterpreting the failed screen. It learns a per-cell causal-role coefficient
+from scalar antithetic cursor observations and multiplies that role by
+within-episode performance innovation
+`|e_(t-1)|-|e_t|`; `kappa=0`, so the branch tests plasticity rather than online
+control. R1 uses only development task seeds 0--2. The complete two-rate
+screen selects `eta=0.1`: all three seed-level ten-check gates pass, final
+success is `98.05%`, `98.83%`, and `99.61%`, sign inversion is
+`0.0651--0.0675`, and learned-role cosine is `0.945--0.973`. The lower
+`eta=0.03` rate retains the mechanism checks but fails final success at
+`36.72--46.09%`.
+
+R2 then evaluates the selected rate once on untouched task seeds 10--15 and
+model seeds 0--4, treating the six tasks--not the 30 models--as independent
+units. Its main task-cluster results are:
+
+| frozen R2 quantity | mean | one-sided 95% relevant bound | gate |
+|:--|--:|--:|:--:|
+| intact final success | 99.531% | lower 99.364% | pass |
+| early-to-late gain | 90.451 points | lower 89.222 | pass |
+| gap over fixed vectorizer | 99.531 points | lower 99.364 | pass |
+| learned-role cosine | 0.9395 | lower 0.9336 | pass |
+| absolute residual-soma correlation | 0.0609 | upper 0.0625 | pass |
+| raw-minus-residual correlation | 0.9365 | lower 0.9350 | pass |
+| surrounding-state balanced accuracy | 54.508% | lower 54.431% | **fail mean >=55%** |
+| decoder-distance residual correlation | 0.1559 | lower 0.1520 | pass |
+| residual outcome balanced accuracy | 47.325% | lower 43.067% | **fail** |
+| residual-minus-soma outcome accuracy | -4.322 points | lower -9.101 | **fail** |
+| causal-role sign inversion | 0.0712; 30/30 positive | lower 0.0703 | pass |
+| velocity-minus-error correlation advantage | 0.6389 | lower 0.6376 | pass |
+| early-residual/late-soma correlation | -0.0132 | lower -0.1786 | **fail** |
+
+All 30 records are finite and pass pairing, neutral-predictor, cursor-cost,
+role-learning, plasticity-lesion, oracle-ceiling, decorrelation, and
+provenance checks. However, the complete frozen R2 gate fails seven aggregate
+population-vectorization/longitudinal checks. It therefore does not establish
+oral-B innovation-guided plasticity under the predeclared joint claim, does
+not raise the reviewer score above 7, and does not unlock oral-A. The strongly
+positive learning, plasticity-lesion, sign-inversion, and velocity-dominance
+results remain useful mechanistic evidence, but they cannot be reported as a
+passed Harnett-signature panel.
+
 ## Frozen endogenous mixed-traffic confirmation (MNIST, d3/w256, 15 epochs)
 
 The 95-run C1 panel crossed five paired model seeds, soma and endogenous top-down traffic, two
diff --git a/REVIEW_SCORECARD.md b/REVIEW_SCORECARD.md
index 4efdf72..483114c 100644
--- a/REVIEW_SCORECARD.md
+++ b/REVIEW_SCORECARD.md
@@ -49,8 +49,9 @@ Every formal result report records:
    but cost `68.4x` ordinary forward-equivalent work.
 3. Innovation was not uniformly beneficial for arbitrary endogenous top-down traffic, so the
    supported mechanism is narrower than the initial claim.
-4. The frozen oral-B screen falsified the desired-velocity and Harnett error-derivative claims.
-   A temporal-difference factorization repairs the sign algebra only; it has no task endpoint.
+4. The temporal-difference recovery solves the task, passes the plasticity lesion, and yields
+   30/30 positive sign inversions, but its untouched R2 panel fails residual outcome advantage and
+   longitudinal prediction. The broader Harnett-like population signature remains unsupported.
 5. The standard-network result inherits reciprocal KP and pays for a paired neutral microphase.
    D4 establishes the innovation operation, not a new credit-transport mechanism or a literal
    cortical implementation.
@@ -81,14 +82,15 @@ Every formal result report records:
 | Dynamic projection D2 | 5 | One frozen 20-epoch record reaches 83.58%, above clean KP's 82.66%, with all stability and cost gates passing | Short single-seed validation cannot establish the full scaling claim |
 | Dynamic projection D3 | 6 | All 19 frozen checks pass at 91.18%, within 0.44/0.08 points of BP/clean KP, with 0.9994 early alignment and 1.326x BP MACs | One validation seed cannot establish robustness |
 | Dynamic projection D4 | 7 | All ten untouched records pass: dynamic 91.584% versus clean KP 91.388%, paired upper deficit bound 0.131 points, early alignment 0.999687, and no invariant failures | Establishes ResNet-20 robustness/noninferiority, not positive depth utility |
-| Oral-B recovery R1/R2 | R1 open | Role/velocity factorization passes mechanics; the two-rate R1 and untouched 6-task-by-5-model R2 gates were frozen before any recovery task endpoint | Only a complete R2 pass can establish innovation-guided plasticity and move 7 to 8 |
-| Oral-A dynamic depth recovery | not opened | A 60-cell ResNet-20/32/56 BP/DFA/clean-KP/dynamic panel, positive-depth-benefit gate, mechanism invariants, and fair cost bounds are frozen before any new endpoint | It is hard-gated on D4 and oral-B R2; only a complete pass can establish standard-depth scaling and move 8 to 9 |
+| Oral-B recovery R1/R2 | failed at R2 | R1 selects eta 0.1 with 98.05% worst-task success; untouched R2 retains 99.53% mean success and 30/30 positive signs but fails outcome-vectorization and longitudinal gates | Score remains 7; the joint oral-B claim is not established |
+| Oral-A dynamic depth recovery | closed | A 60-cell ResNet-20/32/56 BP/DFA/clean-KP/dynamic panel was frozen before any new endpoint | Its oral-B R2 prerequisite failed, so none of the 50 new cells may run |
 | Oral-A A4 | not opened | The prerequisite A3 gate failed | No oral-A confirmation claim is available |
 
 These are conditional reviewer forecasts, not promised scores. A failed stage leaves its negative
 result in the record and can lower the score if it invalidates a current claim. The original
-oral-B branch remains failed and cannot be retroactively reopened by vision results; the new
-plasticity-only recovery must pass its own separately frozen R1 and R2 gates.
+oral-B branch remains failed and cannot be retroactively reopened by vision results. The
+plasticity-only recovery passes R1 but fails its separately frozen R2 joint gate, so oral-A
+remains closed.
 
 ## Evidence-to-score log
 
@@ -121,6 +123,8 @@ plasticity-only recovery must pass its own separately frozen R1 and R2 gates.
 | 2026-07-22 / `32122d0` oral-B recovery freeze | Before any recovery task endpoint, the untouched 30-record R2 protocol, task-cluster uncertainty, original B1/B2 signatures, plasticity lesion, digest binding, and immutable analyzer are executable | 6 → 6 | Removes a preregistration gap but supplies no empirical evidence; R1 is still sealed behind D4 and confirmation seeds remain untouched |
 | 2026-07-22 / `1853620` oral-A recovery freeze | Before any new standard-depth endpoint, a D4-reusing 60-cell ResNet-20/32/56 panel and exact runner/analyzer contract are executable, with BP/DFA/KP controls, positive depth gain, alignment, projection, query, MAC, and memory gates | 6 → 6 | Precommits the final oral claim without bypassing the required sequence; no experiment opens unless D4 reaches 7 and oral-B R2 reaches 8 |
 | 2026-07-23 / `2fed62a` dynamic projection D4 | All ten untouched paired test records pass the frozen gate: dynamic 91.584% versus clean KP 91.388%, clean-minus-dynamic upper bound 0.131 points, 0.999687 early alignment, and zero invariant failures | 6 → 7 | Establishes the strict accept bar with independent ResNet-20 robustness/noninferiority; opens oral-B R1 but does not support added-depth or desired-velocity claims |
+| 2026-07-23 / `1ff7cb7` oral-B recovery R1 | The complete two-rate development grid selects eta 0.1; all 30 seed-level checks pass, with 98.05% worst-task final success, positive sign inversion, and 0.945 minimum role cosine | 7 → 7 | Strong development support for the repaired causal-role/velocity factorization, but R1 cannot change the score and only opens untouched R2 |
+| 2026-07-23 / `03c94a1` oral-B recovery R2 | Thirty untouched records retain 99.53% mean success, 90.45-point gain, 30/30 positive signs, and strong decorrelation, but fail seven population-vectorization/longitudinal checks | 7 → 7 | The recovery fixes learning, causal role, and sign but not the broader Harnett-like signature; oral-B and oral-A close without threshold repair |
 
 Future rows are appended only after an audited frozen stage. A score staying flat is informative:
 engineering, theory exposition, or visualization may make the paper more defensible without
diff --git a/ROADMAP.md b/ROADMAP.md
index d10db78..c40efc5 100644
--- a/ROADMAP.md
+++ b/ROADMAP.md
@@ -20,9 +20,9 @@ not count.
 
 The preregistered oral-B task, structural screen, confirmation seeds, signature thresholds, and
 phase-specific lesion are specified in `ORAL_B.md`. They were frozen while C4 author baselines
-were still running and before any continuous-BCI result was generated. Oral-B confirmation remains
-sequenced after C4; implementation and CPU-only mechanics may be prepared while the accept jobs
-occupy both authorized GPUs.
+were still running and before any continuous-BCI result was generated. Oral-B confirmation was
+sequenced after C4 and the D4 accept gate; the realized original and recovery branches are
+reported below.
 
 **Oral-B development status: failed.** The complete frozen 12-candidate by three-task-seed screen
 found no eligible mechanism. High-learning-rate variants learned the BCI reliably, and several
@@ -32,24 +32,28 @@ preregistered stop rule, environment seeds 10--15 and all confirmation model see
 untouched. This falsifies the current model's claimed emergence of the Harnett derivative signature;
 it cannot be repaired by reporting only its successful learning or decoder results.
 
-**Oral-B recovery R0 status: mechanics passed, task gate sealed.** Reinspection
+**Oral-B recovery R0/R1 status: mechanics and development passed.** Reinspection
 of Fig. 5 and Extended Data Fig. 13 identifies a structural target conflict:
 the failed vectorizer regressed `e_t s_i`, so its role contrast encoded current
 error magnitude rather than the sign of error change. `ORAL_B_RECOVERY.md`
 factorizes a new candidate into a perturbation-learned causal role and the
 within-episode performance innovation `|e_(t-1)|-|e_t|`. Deterministic checks
 recover the role at cosine `0.9963` and change the controlled sign index from
-negative to `+0.100`. This is mechanics only; no BCI endpoint, development
-seed, or confirmation seed is touched. The two-rate, three-task-seed R1
-learning protocol and complete-grid analyzer are now committed, but its runner
-is hard-gated on a complete D4 pass. Confirmation seeds remain sealed.
-
-The untouched recovery confirmation is now also frozen before any R1 task
-endpoint. If and only if R1 selects an eligible rate, R2 runs task seeds
-10--15 by model seeds 0--4 with task-cluster uncertainty, the original B1/B2
-signature thresholds, and a plasticity lesion. A complete R2 pass supports
-innovation-guided plasticity and raises the strict score from 7 to 8; it cannot
-support online control because the recovery fixes `kappa=0`.
+negative to `+0.100`. After D4 passes, the frozen two-rate, three-task-seed R1
+screen selects `eta=0.1`; all 30 seed-level checks pass and worst-task final
+success is `98.05%`. R1 is development evidence and leaves the score at 7.
+
+**Oral-B recovery R2 status: failed; branch closed.** The untouched 6-task by
+5-model confirmation retains `99.53%` mean final success, `90.45` points of
+learning gain, strong fixed-vectorizer and plasticity-lesion margins, mean
+role cosine `0.9395`, positive sign inversion in all 30 records, and a `0.6389`
+velocity-over-error advantage. Residual decorrelation and decoder-distance
+prediction also pass. The joint biological gate fails because
+surrounding-state accuracy is `54.51%` versus the 55% mean threshold, residual
+outcome accuracy is only `47.33%`, residuals trail soma outcome decoding by
+`4.32` points, and longitudinal prediction is `-0.013`. The score remains 7,
+oral-A stays sealed, and no threshold repair or replacement confirmation is
+permitted. Because `kappa=0`, neither R1 nor R2 could support online control.
 
 ## Frozen accept claims and gates
 
@@ -585,6 +589,10 @@ strict reviewer score rises from 6 to 7, establishing the accept bar and
 opening oral-B recovery R1.  Oral-A remains sealed until the separately frozen
 oral-B R2 confirmation passes.
 
+R1 subsequently passes and selects `eta=0.1`, but the complete untouched R2
+gate fails the outcome-vectorization and longitudinal signatures. Oral-A
+therefore remains sealed permanently under this frozen sequence.
+
 Prepare convolutional local-update primitives and ResNet-20/32/56 protocols early. Queue frozen
 runs opportunistically on authorized idle GPUs. Because BurstCCN already reports CIFAR-10 and
 ImageNet scaling, dataset scale alone is not novel. The oral-level target is a memorable joint