results: close oral B signature screen

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@@ -120,6 +120,56 @@ result therefore depends on subtracting the soma-predictable component, not mere
 normalizing an oversized update. This is the most direct evidence that the Harnett-specific
 innovation operation is algorithmically necessary.
 
+## Continuous-BCI Harnett-signature screen (development only)
+
+The oral-B protocol was committed before any result and then run as a complete
+12-candidate by three-environment grid at clean revision `b4458cd`. It crossed
+scalar error versus error-plus-within-episode velocity, online-control gains
+`0`, `0.1`, and `0.3`, and forward rates `0.01` and `0.03`. Every result paired
+intact, fixed-vectorizer, online-lesion, plasticity-lesion, and both-lesion
+conditions on identical continuous trajectories. Confirmation environment
+seeds 10--15 were never evaluated.
+
+| feedback | kappa | eta | worst final success | worst sign-inversion | eligible |
+|:--|--:|--:|--:|--:|:--:|
+| error | 0.0 | 0.01 | 0.156 | -0.0038 | no |
+| error | 0.0 | 0.03 | 0.988 | -0.0018 | no |
+| error | 0.1 | 0.01 | 0.141 | -0.0035 | no |
+| error | 0.1 | 0.03 | 0.988 | -0.0018 | no |
+| error | 0.3 | 0.01 | 0.102 | -0.0032 | no |
+| error | 0.3 | 0.03 | 0.984 | -0.0017 | no |
+| error + velocity | 0.0 | 0.01 | 0.258 | -0.0089 | no |
+| error + velocity | 0.0 | 0.03 | 0.996 | -0.0071 | no |
+| error + velocity | 0.1 | 0.01 | 0.219 | -0.0087 | no |
+| error + velocity | 0.1 | 0.03 | 0.996 | -0.0069 | no |
+| error + velocity | 0.3 | 0.01 | 0.156 | -0.0085 | no |
+| error + velocity | 0.3 | 0.03 | 0.992 | -0.0068 | no |
+
+The screen demonstrates a sharp dissociation. At `eta=0.03`, all structural
+variants solve the task (worst-environment final success 98.4--99.6%). Raw
+apical activity is almost completely soma-correlated (mean absolute
+correlation `0.999`), whereas residualization removes most of that dependence
+(raw-minus-residual correlation gap `0.880`). Surrounding-network activity
+decodes residual sign at mean balanced accuracy `0.727`, and early residuals
+strongly predict later activity changes (`r=0.860` on average). Plasticity
+lesion eliminates learning gain in every run.
+
+These positive signatures do **not** pass oral B. All 36 trajectories have a
+negative causal-role sign-inversion index (range `-0.0089` to `-0.0011`), and
+role-aligned residuals are more associated with instantaneous error magnitude
+than with signed error velocity in every run. Acute removal of online control
+after intact training drops success by only `1.03` points on average (maximum
+`5.08`), so the desired-velocity interpretation is not supported. Across the
+36 runs, the frozen per-run checks failed 36 times for sign inversion, 30 for
+the strict residual-soma threshold, 6 for the fixed-vectorizer margin, and 5
+for learning gain. The preregistered selector therefore returns
+`failed_no_eligible_candidate`, and oral-B confirmation is prohibited.
+
+This negative result prevents a misleading inference: successful local
+learning, outcome decoding, and residual decorrelation do not imply the
+cell-role-specific error-derivative signature observed by Harnett and
+colleagues.
+
 ## Frozen endogenous mixed-traffic confirmation (MNIST, d3/w256, 15 epochs)
 
 The 95-run C1 panel crossed five paired model seeds, soma and endogenous top-down traffic, two